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4 questions
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  • Q1
    Oncogenes are mutated forms of genes. Oncogenes can transform a cell into a tumor cell. Some tumor cells are benign, while others are malignant. How does the presence of an oncogene lead to the formation of a tumor?
    ATP production is inhibited
    Cell division is unregulated
    Somatic cell growth is inhibited
    Antibody activity is unregulated
    B.5.D: Diseases
  • Q2
    Cells typically respond to DNA damage in three ways: by ceasing to grow and divide until the damage is repaired, by permanently ceasing to grow and divide, or by dying. In 2010 a group of scientists reported that a certain kind of immune reaction can cause DNA damage that leads to a fourth response. DNA damage can turn off genes involved in cell-signaling pathways. Turning off these genes can cause less-mature cells to divide too rapidly, often leading to the development of —
    cardiovascular disease
    B.5.D: Diseases
  • Q3
    Atherosclerosis is a disease that obstructs blood flow and, therefore, oxygen supply to target organs. A major component of atherosclerosis is the excessive reproduction of smooth muscle cells of the blood vessels. Certain drugs may have the potential to reverse or prevent the unregulated reproduction of the diseased blood-vessel cells. Which of these processes is the most likely target of these drugs?
    DNA transcription
    Erythroblast differentiation
    Cellular respiration
    Cell division
    B.5.D: Diseases
  • Q4
    The diagram below represents the cell cycle. When cells leave the cell cycle, they exit during G1 phase and then enter G0 phase, a resting period. Most normal cells can leave G0 phase and reenter the cell cycle at G1 phase before entering S phase. Cancer cells are different because they cannot enter G0 phase and are likely to do which of the following?
    Question Image
    Die after completing mitosis
    Mutate during G1 phase
    Fail to complete S phase
    Repeat the cell cycle continuously
    B.5.D: Diseases

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