Area of cone | Quizalize

Surface Area of Cones, Cylinders & Spheres

Surface area of a frustum of a cone

Surface Area & Volume of Cones

The surface area of a cone

12.3 - Surface Area of Pyramids and Cones

3. Volcanoes are often found at meeting points of “tectonic plates”. These plates are pieces of the Earth’s surface that fit together just like a jigsaw puzzle. 4. Volcanoes can also occur over “mantle plumes”. Ever heard of mantle plumes? They’re super-hot areas of rock inside the Earth! Did you know that we have a FREE downloadable Volcanoes of Ecuador primary resource? Great for teachers, homeschoolers and parents alike! 5. Approximately 350 million people live within “danger range” of an active volcano. That means that around one in 20 people live in an area at risk of volcanic activity. Volcano facts 6. Volcanoes are classified as active, dormant or extinct. This refers to the amount of volcanic activity. “Active” means there’s regular activity, “dormant” means there’s been recent activity but the volcano is currently quiet, and “extinct” means it’s been so long since the last eruption that it’s unlikely to ever erupt again. 7. Volcanoes can be a variety of shapes. These geographical wonders come in various shapes and sizes, but there are two main types – composite volcanoes, which are cone-shaped with steep slopes, and shield volcanoes, which are wide with gentle slopes. 8. Magma and lava are two different things! Magma is the name given to hot liquid rock inside a volcano. Once it leaves the volcano, it’s known as lava. Psst! Make sure you download our awesome volcano facts infographic – show your friends, family, or stick it on your wall!

Plăcile de rețea (Network Interface Cards-NICs) conectează un echipament la rețea. Plăcile de rețea Ethernet sunt folosite pentru o conexiune cablată, în timp ce plăcile de rețea WLAN (Wireless Local Area Network) sunt folosite pentru wireless. Un echipament cu utilizator final ar putea include unul sau ambele tipuri de plăci de rețea. De exemplu, o imprimantă de rețea poate avea doar o placă de rețea Ethernet, așadar, trebuie să se conecteze la rețea printr-un cablu Ethernet. Alte echipamente, precum tabletele sau telefoanele pot conține o placă de rețea WLAN și trebuie să folosească o conexiune wireless. Layer-ul Fizic Layer-ul fizic de la OSI furnizează mijloacele de transport a biților care realizează un frame de data link în mediul de rețea. Acest layer acceptă un frame complet de la layer-ul data link și îl codifică sub forma unor serii de semnale care sunt transmise în mediul local. Biții codificați care comprimă un frame sunt primiți fie de un echipament final, fie de unul intermediar. Procesul prin care trec datele de la nodul sursă la nodus destinație este: • Datele utilizatorului sunt segmentate de layer-ul transport, plasate în pachete de către layer-ul rețea, iar apoi încapsulate sub formă de frame-uri de către layer-ul data link. • Layer-ul fizic codifică frame-urile și creează semnalele undelor electrice, optice și radio care reprezintă biții în fiecare frame. • Aceste semnale sunt trimise în mediu pe rând. • Nodul destinație preia aceste semnale individuale de la nivelul fizic, le transformă în reprezentare binară și transmit biții la nivelul superior, data link, sub forma unui frame. Mediul layer-ului Fizic Există trei forme de bază ale mediului de rețea. Layer-ul fizic produce reprezentarea și gruparea biților pentru fiecare mediu, după cum urmează: • Cablu de cupru: Semnalele sunt modele ale pulsurilor electrice. • Cablul cu fibră optică: Semnalele sunt modele de lumină. • Wireless: Semnalele sunt modele ale transmisiunilor cu microunde. Figura afișează exemple de semnalizare pentru cupru, fibră optică și wireless. Pentru a activa interoperabilitatea layer-ului fizic, toate aspectele acestor funcții sunt guvernate de organizațiile de standardizare. Standardele Layer-ului Fizic Protocoalele și operațiile layerelor superioare din OSI sunt efectuate în software proiectat de ingineri și oameni de știință. De exemplu, serviciile și protocoalele din suita TCP/IP sunt definite de Internet Engineering Task Force (IETF) în RFC-uri așa cum se arată în Figura 1. Layer-ul fizic constă în circuite electronice, mediu și conectori dezvoltați de ingineri. Așadar, este corespunzător ca standardele care guvernează acest hardware să fie definite de orgnizațiile relevante din domeniul ingineriei și electronicii. Există mai multe organizații naționale și internaționale diferite, organizații guvernamentale de reglementare și companii private implicate în stabilirea și menținerea standardelor layer-ului fizic. De exemplu, hardware-ul layer-ului fizic, mediul, codificarea și standardele de semnalizare sunt definite și guvernate de către: • International Organization for Standardization (ISO) • Telecommunications Industry Association/Electronic Industries Association (TIA/EIA) • International Telecommunication Union (ITU) • American National Standards Institute (ANSI) • Institute of Electrical and Electronics Engineers (IEEE) • Autoritățile de reglementare națională a telecomunicațiilor, inclusiv Federal Communication Commission (FCC) din USA și European Telecommunications Standards Institute (ESTI) Pe lângă acestea, există grupuri de standardizare a cablărilor regionale precum CSA (Canadian Standards Association), CENELEC (European Committee for Electrotechnical Standardization) și JSA/JSI (Japanese Standards Association), care dezvoltă specificații locale. Figura 2 listează contribuitorii principali și câteva standarde relevante ale layer-ului fizic.

[t comes from the GREEK name "Epilepsia" which means "taking hold of or seizing". - It is a disorder characterized by: recurrent seizures. SEIZURES R ectment transient attacks of: R epresent: R esult from: ASSOCIATED WITH: somatic, psychic, or, autonomic clinical featmes. clinical features of abnormally hyperexcitable cortical neurons. paroxvsmal and excessive electrical neuronal discharges. EEG changes & may be disturbance of consciousness. same causes of convulsions 1. Idiopathic epile~ • It is the commonest cause. no cause can be detected ( 65 % ) • It may be associated with positive family history in some cases. • It starts in the l st & 2nd decades in the form of: -- Grand ma! epilepsy. Petit mal epilepsy. Myoclonic epilepsy. Atonic seizures. 2. Secondary epilepsy A. Local causes in the brain: l. Congenital: 2. Traumatic: cerebral palsy. a cause can be detected cerebral contusion or laceration. 3. Inflammatory: 4. Neoplastic: 5. Degenerative: 6. Vascular: encephalitis, brain tumours. mening1t1s, presenile dementia. brain abscess. stroke (especially hemon-hagic), hypertensive encephalopathy. B. General causes with secondary effects on the brain: I. Toxic: 2. Iatrogenic: 3. Metabolic: 4. Endocrinal: 5. Organ failure: 6. Heart disease: 7. Nutritional: - Alcohol, cocaine, lead. - Lidocaine, INH. - j glucose & ! glucose. - Hypoparathyroidism. - Hepatic failme. - Adam's Stoke's attacks. - Pellagra. - Botulism, tetanus. - Ambilhar, Amphetamine, Aminophylline. - j Ca & ! Ca. - Hype1thyroid crisis. - Renal failure. - Fallot's tetralogy. - j Na & ! Na. - Vitamin B6 deficiency. 8. Physical: 9. HYSTERICAL. - High fevers. - Heat stroke. 136 137 CLINICAL PICTURE 1. GENERALISED SEIZURES " Excessive electrical discharges from cortical neurons in BOTH hemispheres simultaneously " I. II. 1. Grand Mal Epile~: 1. Pre-ictal stage "attacks of tonic-clonic convulsions " (aura) It is a warning sign of a coming attack. It may be: • Somatic: • Psychic: • Autonomic: 2. Ictal stage Myoclonus, Hallucinations. Tachycardia, (seizure) Sudden loss of consciousness: Parasthesias. Sweating. for seconds to minutes. -- Tonic phase (few seconds) o The UL & LL: o o o o The HEAD: The JAWS: CYANOSIS: are extended. is retracted to one side & the eye balls rolled up. are firmly clenched, with biting of the TONGUE. due to impaired respiration. There may be incontinence of urine. Clonic phase (few minutes) o The UL & LL: o The HEAD: 3. Post-ictal stage - It may be: • Somatic: • Psychic: • Autonomic: Drug of choice: contract & relax repeatedly & rapidly. jerks forcibly. (sequelae) Todd's paralysis(< 24 hours, due to neuronal exhaustion). Confusion. Vomiting. Carbamazepine (Tegretol) or Phenytoin (Epanutin) Petit Mal Epilepsy: "attacks of loss of consciousness " " Absence " It starts in childhood & improves at puberty & usually disappears at the age of 20. 2. It is NOT PRECEEDED by aura & NOT FOLLOWED by sequelae. 3. It is usually PRECIPITATED by: hyperventilation 4. It is characterized by: or photic stimulation. sudden loss of consciousness of short duration (few seconds). 5. It may be associated with: • High frequency ( 50 attacks / day). • Falling to the ground without warning. • Jerky movements of the head & UL Drug of choice: (myoclonic petit mal). Valproate (Depakine) or Succinimide (Zarontin) 137 138 Ill. M oclonic Seizures: "attacks of involuntary clonic movements " - It is characterized by: sudden, jerky, shock-like INVOLUNTARY muscle contraction. • The jerks are bilateral contractions, mainly of the shoulders and arms. • However, some patients repmtjerking in the lower limbs, trunk, or head. - It may be of 2 types: - Occurs singly • Simple: • As a pait of: I Drug of choice: IV. Atonic seizures: (no loss of consciousness). - Grand mal epilepsy (aura). - Petit mal epilepsy. Valproate (Depakine) or Clonazepam (Rivotril) I - Transient attacks of brief loss of postural tone, often resulting in falls and injuries. 2. PARTIAL SEIZURES "Excessive electrical discharges from cmtical neurons in a ce1tain area in ONE hemisphere" A. Simple seizures: " No disturbance in consciousness " - The CP depends on the site of the hyperexcitable neurones in the cerebral cortex, whether in: "Motor area or Senso,y areas". 1. Motor fits: • Focal fits: • Motor jacksonian fits: 2. General Sensory fits: • Focal fits: • Sensory jacksonian fits: 3. Special Senso1y fits: • Visual hallucinations: • Auditory hallucinations: • Olfactory hallucinations: B. Complex seizures: - SITE: movement of part of a limb or the whole limb. movement of one side of the body (see before). parasthesia of part of a limb or the whole limb. parasthesia of one side of the body (see before). irritation of the visual sensory area. irritation of the auditory sensory area. initation of the uncus. " disturbance in consciousness " The hyperexcitable neurons are in the Temporal lobe "Temporal lobe epilepsy". - DURATION: The seizure lasts few seconds to few minutes. - The seizure starts with A ura, followed by A bsence, Automatism, Amnesia: 1. 2. 3. 4. A ura: A bsence: Automatism: A mnesia: Olfactory hallucinations, Deja-vu phenomenon, Sensation of fear. Absent patient with staring eyes (with no response to conversation). Involuntary Purposeless acts: motor ( eg, lip smacking, chewing) or verbal. No recalling of the seizure. 138 139 3. PARTIAL SEIZURES ~ GENERALISED SEIZURES " Partial seizures may spread to involve the whole brain .- secondarily generalised seizures " . HY-sterical epilepsY • Usually: • The cause: • Incidence: young neurotic Sj2 . psychological & there is no organic lesion. usually occurs in the presence of people. • It is associated with: • EEG: • It is not associated with: normal. • Missed ttt. • Menses. • Alkalosis. anxiety, palpitaion & hyperventilation. tongue biting or incontinence of urine. • Alcohol use & Drug abuse ( e.g. cocaine ). • S timulation by photons & Hyperventilation. • S leep deprivation & Stress & sudden withdrawal of antiepileptic drngs. INVESTIGATIONS 1. EEG: • It is the most specific test for epilepsy because it records the electrical activity of the brain. • It shows specific pattern: 2. LOCAL INVESTIGATIONS: "Epilepsy waves". "CT & MRI of the brain" • To identify or exclude a LOCAL CAUSE of seizures in the brain. 3. GENERAL INVESTIGATIONS: "Laboratory investigations" • To search for a GENERAL CAUSE of seizures, e.g. blood glucose. 139 140 TREATMENT A. General Measures: 1. 2. Moderation of the patient's physical activity. A void the precipitating factors ( Alcohol, hyperventilation, photic stimulation ...... ). 3. A ketogenic diet is encouraged because it will induce acidosis: - Acidosis is beneficial as it raises the threshold of stimulation of the brain cells. B. Specific Treatment: 2. 1. Treatment of the cause in secondary epilepsy. Anti-epileptic drugs: a) Always sta1t with one drug, then add another drug if there is no response. b) Always stop the drugs ONLY if: • The patient stays free of symptoms for at least 2 years. • The patient has a normal EEG. 3. Side effects of Anti-epileptic drugs: I . Skin rash. 2. 3. Bone marrow depression. Ataxia. Drug 1. Barbiturates (Pbenonobarbitone) 2. Hydantoin (Epanutin) 3. Carbamazepine 4. Clonazepam 5. Valproate 6. Succinamide ANTI-EPILEPTIC DRUGS NEW ANTI-EPILEPTIC DRUGS - These drugs are new dtugs that may be used in resistant seizures. 1. Lamotrigine: 200 - 400 mg/ day. 2. Felbamate: 3. Gabapentin: 400- 800 mg/ day. 600 - 1200 mg/ day. \ " General rules for use ": Dose 100-600 mg I day 100-600 mg / day 200-600 mg I day 2-6 mg I day 500-1500 mg I day 500-1000 mg / day Best indicated - Broad spectrum. - Not for petit mal. - Grand mal. - Motor Jacksonian fits. - Grand mal. - Motor Jacksonian fits. - Complex seizures. - Not for petit ma!. - Myoclonic. - Grand mat. - Broad spectrum. - Petit mat. 140 141 STATUS EPILEPTICUS DEFINITION - A medical emergency: 1. Repeated attacks of generalized convulsions, with lack of recove,y of consciousness, 2. Persistent attack of seizure lasting for at least 30 minutes. OR, - If the convulsions are not stopped rapidly, coma deepens & death may occur due to: heart failure or respiratory failure or brain damage or hyperpyrexia. - The most common causes are: sudden withdrawal of anti-epileptic drugs & stroke. TREATMENT A. General Measures: l. Take care of: " ABC " • Place the patient on the ground, to guard against falling from bed. • Mouth gag & 02 inhalation ( endo-tracheal intubation may be needed). • Record the vital signs regularly. 2. Take a sample of: - Venous blood: for the level of: - A.tierial blood: for the level of: 3. a nti-epileptic drugs, a lcohol. pH, p0 2, pC02, HC0 3. Give cerebral dehydrating measures: e.g. Frusemide, cone. Mannitol, Dexamethazone. B. Specific Treatment: - Phenytoin with diazepam (or clonazepam) immediately: 1. Phenytoin: 2. Diazepam: Clonazepam: seizures recur: 15 mg I Kg slow infusion. 5 mg slowly IV, to be repeated after 5 minutes if seizures recur: maximum dose: 20 mg. OR: 2 mg slowly IV, to be repeated after 5 minutes if maximum dose: 6 mg. - If seizures persist after 20 min. of Phenytoin & diazepam: 3. PHENOBARBITONE: - In resistant cases: 200 mg infusion. 4. GENERAL ANAESTHESIA: may be used.

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